Introduction: Vertebrabasal Insufficiency is clinical syndrome whose characteristics is that mainly start suddenly with vertigo followed by nausea and vomiting, unsafe walk and standing. Pathophysiologicaly, Vertebrabasal Insufficiency is transitory ischematic attack in posterior - VB flow with caracteristis like disorder of function of brain tree, cerebellum, including part of cerebrum too. Heart Arrhythmia are changes in autonomy or conducting of impulses.Source of those changes may be all parts of heart conduction system and there are number of causes, which result arrhythmia, like local heart diseases (born anomalies, inflammatory processcs, degenerative diseases),up to systematic (anemia, hypertension, different metabolic disorders). Objectives: The objective of this work is effect of arrhythmia as etiology factors in case VB Insufficiency occurs in middle life age. Material and Methods: Work analysed a group of 50 subjects with clinical picture of VB Insufficiency, age of 40 to 60 years and control group of 50 subjects without symptoms of VB Insufficiency of 40 to 60 years of age too. Average age in both groups was 49. Following method of transcranial Doppler sonography used with all subjects in experiment group, lower value of middle speed of blood flow in one or more arteries of VB flow were found. In group of subjects with symptoms of VB Insufficiency arrhythmia was confirmed (auscultatory and by ECG) with 7 subjects, out which in one case bradycadr and rest of 6 subjects had tachycard, but in control group arrhythmia was confirmed with one subject only. Nine subjects in experiment group had established ishchemic changes of ECG and only one subject in control group. Hypertension artherialis was established with 19 subjects in experiment group and with 2 in control group, anemia was notified with one subject only in experiment group.
Conclusion: Heart arrhythmia as etiology factor takes significant place in pathogenesis of VB Insufficiency in middle life age.It would be possible to prevent attacks of VB Insufficiency by timely disclosure and treatment of causes which may result to arrhythmia, before all hypertensy, anemia, inflammatory, tocsic, degenerative and metabolic, as well as disorders of sympatic and parasympatic system.